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During an asthma exacerbation and in the case of uncontrolled asthma, the airway environment becomes more acidic and oxidizing that leads to the release of NO from S-nitrosoglutathione (GSN), which leads to high levels of exhaled NO [2]

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Additionally, ROS-induced cysteine glutathionylation and its modulation have been shown to be the key regulators of IL-1beta bioactivity by preventing the irreversible ROS-elicited deactivation [111]