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Modulation of the GLP-1 - K ATP pathway by AMP kinase, NO and ACh The ability of GLP-1 released by RPc to relax cardiac pericytes and increase coronary blood flow presumably depends on the activation of K ATP channels in the surface membrane of the pericytes, which will hyperpolarise the pericytes and decrease voltage-gated Ca 2+ entry

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Studies have also linked the drugs to a host of other benefits, including a lower risk of heart attacks, strokes, dementia, clotting disorders, addiction and sleep apnea

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Both peptides mimic endogenous growth-hormone-releasing hormone (GHRH), the hypothalamic signal that tells pituitary somatotrophs to secrete growth hormone