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The goal is to understand why inflammatory signaling is elevated and whether modifiable drivers are present: visceral fat, periodontal disease, poor sleep, alcohol, insulin resistance, autoimmune activity, environmental exposure, chronic infection, or unresolved cardiometabolic risk

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Additionally, interactions between therapeutic peptides and endogenous luminal peptides or proteins represent an underexplored source of variability that can influence both permeability and stability (Moughan et al., 2014)

boulardii supplementation was associated with decreased serum levels of C-reactive protein and IL-18, along with attenuation of systemic inflammatory responses [78, 79]

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