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Vignoli A, Ghini V, Meoni G, Licari C, Takis PG, Tenori L, Turano P, Luchinat C

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232,233 In addition to the inactivating mutation of Wnt/-catenin pathway components, abnormal secretion of Wnt ligands in many tumours can also lead to overactivation of the Wnt/-catenin pathway (DKK1, Wnt3a, and Wnt5a)

GLP-1 RAs have been demonstrated to effectively support cardiomyocyte survival by addressing a number of key mechanisms, including impaired mitochondrial Ca 2 processing, abnormal energy metabolism, structural abnormalities, increased oxidative stress, and ER stress mediated by NF-B (Fig