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The role of PDGFR- activation in acquired resistance to IGF-1R blockade in preclinical models of rhabdomyosarcoma

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Together, excessive and reduced production of neuro-toxic and -protective kynurenines, respectively, may exacerbate excitotoxicity and neuroinflammation, establishing a feed-forward cycle of KP dysregulation, 5-HT depletion, HPA axis hyperactivity, and increased vulnerability to MDD

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These results establish the HAGSHPVA scaffold as a redox-active, ECM mimetic nanofibrous platform with strong potential for next-generation antioxidant-enriched wound-healing applications