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Blocking the HGF/c-Met signaling pathway suppresses PNI in vivo To further understand the crosstalk between pancreatic cancer cells and nerves and whether blocking the HGF/c-Met signaling pathway inhibits PNI in pancreatic cancer in vivo, a sciatic nerve invasion model was established by injecting BxPC-3 cells (sh NC vs

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Normal ATDC5 cells were seeded in a 96-well plate (1 10 4 cells per well) and cultured with DMEM containing 10% FBS for 24 h at 37

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This parallel makes Dihexa an interesting crossover compound for researchers investigating whether repair mechanisms in the periphery (tendons, gut) share common signaling logic with repair mechanisms in the brain (synapses, dendrites)