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Oxidative stress in helicobacter pylori infection: Does supplementation with vitamins c and e increase the eradication rate

Mechanisms include: Restoration of endothelial nitric oxide synthase (eNOS) Upregulation of VEGF and FGF-2 (fibroblast growth factor 2) Reduction of oxidative stress and cytokine-induced apoptosis BPC-157 preserved endothelium-dependent vasodilation via upregulation of eNOS and suppression of vascular inflammation. Gwyer Findlay et al., Peptides 4

How can clinicians best support their patients taking GLP-1s to practically apply these nutrition and lifestyle interventions into their daily lives

doi:10.1248/bpb.32.651

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