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Furthermore, the excessive accumulation of ROS not only disrupts the process of melanin synthesis, but also destabilises, synthesises and disrupts the circulation of lipids in melanocytes, resulting in damage to the mitochondrial electron transport chain and increased ROS production, creating a vicious cycle and destroying melanocytes [19]

Actually the structures of U46C GPx4 mutant and rhGPx3 mutant are quite similar (Figure 4a) although their sequence identity is only 34%

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Others have reported that IC/BPS patients with Hunner lesions are older and more likely to have other comorbid pain syndromes, and that those patients without Hunner lesions more likely to have pelvic floor dysfunction and myofascial pain (