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The pathological processes described above are often initiated in schizophrenia by a breach of the intestinal barrier, creating a conduit for gut-derived immune activation

The results obtained showed that metabolic treatment has very limited toxicity as no patient experienced major side effects

Moreover, THBS1 can mitigate the menstruation-driven inflammatory response by binding to cell-surface receptors (such as a4b1 integrin) of inflammatory cells, thereby restoring the overall immune homeostasis when proceeding to the next stages of the menstrual cycle [18]

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10.3389/fimmu.2015.00223 105 De VriesY