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Critically, these biochemical alterations were corroborated by distinct histopathological and immunohistochemical evidence of thyroid damage, including follicular cell atrophy, vacuolization, and strong immunoreactivity for iNOS, eNOS, and PCNA

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doi: 10.1016/j.trsl.2014.02.003 103 NilssonEKingSEMcBirneyMKubsadDPappalardoMBeckDet al

Gelinas R, Mailleux F, Dontaine J, Bultot L, Demeulder B, Ginion A, Daskalopoulos EP, Esfahani H, Dubois-Deruy E, Lauzier B, Gauthier C, Olson AK, Bouchard B, Des Rosiers C, Viollet B, Sakamoto K, Balligand JL, Vanoverschelde JL, Beauloye C, Horman S, Bertrand L (2018) AMPK activation counteracts cardiac hypertrophy by reducing O-GlcNAcylation