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Beyond the typical regulatory pathways mentioned above, ferroptosis is modulated by many regulators, such as p53, nuclear factor erythroid 2-related factor 2 (Nrf2) and AMP-activated protein kinase (AMPK)[37-39], among which p53 gets a lot of attention due to its controversial role in ferroptosis

GCL is a heterodimeric enzyme composed of a glutamate-cysteine ligase catalytic subunit (GCLC), which confers enzymatic activity, and a glutamate-cysteine ligase modifier subunit (GCLM), which enhances GCLC efficiency and attenuates feedback inhibition by GSH

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