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The authors of these studies therefore concluded that the observed proinflammatory phenotype is likely mediated by increased TGF/NOX/ROS signaling within the HUVECs 23,30

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CXC chemokines,like CXCL1, CXCL2, CXCL5, CXCL8, CXCR1, and CXCR2, generally, are involved in neutrophil chemotaxis (99), but CXCL10 and CXCL13 promote effector T cells and B cells recruitment into the joint, respectively (100, 101)

R., Li, W., Wang, H., Yuan, F., Xu, R., Chariker, J