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[11] Pickart L, Margolina A
It has been demonstrated that increasing ROS could easily result in dysfunctions of proliferation or differentiation in AML, and could be an effective strategy for overcoming drug resistance in leukemia [21]
These converge on two outcomes critical to capillary formation: endothelial cell proliferation (driven primarily by ERK1/2 activation) and endothelial cell survival and migration (driven primarily by Akt phosphorylation)
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