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Estradiol, brain insulin sensitivity and resistance The activation of estrogenic pathways exerts a neuroprotective effect in the CNS through four different mechanisms [reviewed in (53)]

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Methylation pathways become saturated, and excess methionine is converted to homocysteine, which may contribute to cardiovascular risk if B-vitamin cofactors (B6, B12, folate) aren't adequate

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Conversely, (PHA + MSG), (SE + MSG), (AST + MSG), (SE + AST + MSG) and (COMB + MSG) groups both obviously downregulated the gene expression levels of PERK (by 27.7%, 51.8%, 55.2%, 70.2% and 83.2%, respectively) (Fig