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In conclusion, MASLD pathogenesis involves complex interactions among multiple metabolic pathways, including dysregulated lipid metabolism, insulin resistance, inflammation, and oxidative stress

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The condition often develops insidiously, with patients initially attributing symptoms to expected side effects before suddenly deteriorating and requiring emergency intervention

It is known that mTORC2 phosphorylates Akt and appears to regulate mainly cell proliferation and cell survival [110] (Figure 4) The effect growth factors have on mTORC1 is mediated through phosphatidylinositol-3,4,5-triphosphate kinase (PI3K) activation, the subsequent activation of phosphoinositide-dependent kinase (PDK1) and Akt