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Therapeutic strategies to boost levels of protective antiproteases such as A1AT in the lung remain an attractive research strategy to limit the damage from excess protease activity

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An SSRI makes serotonin and norepinephrine reuptake into the presynaptic neuron less likely, allowing these extra neurotransmitters in the synaps to transmit their signal to the postsynaptic neuron for longer 16,20,21