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Specific deletion of A2BAR in alveolar epithelial cells, in comparison to myeloid and endothelial deletions, significantly attenuated pulmonary inflammation and edema ( 2 O 2 )-induced AEC injury revealed lower AEC apoptosis rates with A2BAR activation, with the mechanism of action via suppression of p38 and ERK1/2-mediated mitochondrial apoptosis pathway ( Adenosine signaling is also crucial in endothelial barrier function during acute lung inflammation
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