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The anti-inflammatory actions of GCs are mediated through transrepression , a process in which the ligand-activated GR, often as a monomer, interacts with and inhibits the activity of pro-inflammatory transcription factors, such as nuclear factor-kappa B (NF-B) and activator protein-1 [23]

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These still preliminary observations open mechanistic hypotheses about possible central modulation complementary to peripheral effects

Moreover, our data showed a close association between the net placental release of estradiol and progesterone into the maternal circulation in healthy pregnancies