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Failure to accumulate glycogen because of inanition or abnormal glycogen retention may result from treatment-induced metabolic perturbations

Heliyon 10:e30426

Mechanism in research literature The mechanistic anchor for ipamorelin is selective agonism at the growth-hormone-secretagogue receptor type 1a (GHSR-1a), the same receptor through which the endogenous hormone ghrelin signals

87 HSCs are mainly stimulated to produce hepatocyte growth factor by interleukin-6 (IL-6) trans-signaling rather than through IL-6 signaling following PHx, which indicates the crucial role of soluble IL-6 receptor (sIL-6R) in liver repair