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Dysbiosis and intestinal permeability may increase the delivery of endotoxin, ammonia, aldehydes, phenols and other microbial metabolites to the liver through the portal circulation

PLIC proteins or ubiquilins regulate autophagy-dependent cell survival during nutrient starvation

These findings collectively emphasize that gut dysbiosis, increased intestinal permeability, and even gutlung microbial translocation can amplify neuroinflammation and worsen ischemic stroke outcomes, underscoring the need to consider the gutbrainlung axis as a potential target for improving stroke prognosis

If recovery improves and movement becomes easier, physical activity often increases naturally

Zhang D, Xi Y, Boffa DJ, Liu Y, Nogueira LM