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Ta1 also stimulates IL-2 receptor expression and enhances lymphokine-activated killer cell activity

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This agent is very promising for future clinical use since it perturbs the self-sustaining activation loop of JNK, instead of inhibiting JNK directly, and therefore would not be expected to inhibit the possible protective/beneficial effects of transient JNK activation in AILI [5]

10.1016/j.psyneuen.2009.09.017 Psychoneuroendocrinology 32 GreenoC