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Structurally it is based on the full 44-amino-acid growth-hormone-releasing factor sequence (hGRF 1-44) with a modification that protects it from rapid enzymatic breakdown, extending its usefulness compared with native GHRH

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DIM and I3C enhance the effects of anti-estrogen treatments by acting on AhR-mediated transcription, changing the epigenetics of estrogen-responsive genes, and suppressing ER signalling (199)

Research remains exploratory, focusing on cellular communication, pathway duration, and system-wide signaling responses rather than clinical or physiological outcomes

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