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In the present study, we compared the impact on lung function and immune response of chronic exposure (either 3 or 6 months) to ECA with that of conventional cigarette (3R4F) smoke (Fig

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To explain the discrepancy in the results, it was proposed that the deleterious effect of smoking on AD may have been masked by the fact that subjects who would have subsequently developed AD had either died or left the study due to other co-morbidities 24,25

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