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It was discovered that intracerebroventricular pre-treatment with Ca-EDTA significantly attenuated the ischemia-induced down-regulation of GluR2 mRNA and protein expression in the CA1 hippocampal subfield

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Bergmann et al

View in: Pubmed Induction of T cell clonal anergy results in resistance, whereas CD28-mediated costimulation primes for susceptibility to Fas- and Bax-mediated programmed cell death

The reduction in A plaques in the hippocampus and cortex was significant (25.6% and 31.5%, respectively when compared to control, see Fig