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Considering that nucleoplasmic -catenin did not appear to be significantly influenced, our data provide ostensible evidence that sarcoplasmic -catenin may be playing a key regulatory role in encouraging ARandrogen interactions or activating the AR in an androgen-independent manner

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Documentation should address contraindications to alternative treatments, expected clinical outcomes, and monitoring protocols

10.1007/978-94-007-2199-9_18 39 FenechM

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