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results in disturbances in the homeostatic redox state of cells

Emerging evidence indicates that exercise can suppress ferroptosis directly or indirectly by regulating iron metabolism, oxidative stress, and exerkine expression, thereby conferring neuroprotection

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Research from the American Journal of Physiology (2016) demonstrated that glycine maintains Akt-mTOR-FOXO1 signaling, the anabolic cascade that drives muscle protein synthesis, while simultaneously suppressing TLR4 and NOD2 signaling pathways associated with inflammation-driven muscle breakdown